APP belongs to a protein family that includes APP-like isoforms (APLP-1 and APLP-2 in mammals), which lack the Ab sequence. There appears to be some functional redundancy between APP and its isoforms in mammals, as APLP-1 or APLP-2 can partially compensate for the loss of the APP gene in mouse models. Triple mutants involving APP, APLP-1 and APLP-2 are lethal.
InterPro Domain Architecture
InterPro Entry |
Signatures |
Graphical Match |
Method Name |
IPR002223 |
G3DSA:4.10.410.10 |
|
Prot_inh_Kunz-m |
IPR002223 |
PD000222 |
|
Prot_inh_Kunz-m |
IPR002223 |
PF00014 |
Kunitz_BPTI |
|
IPR002223 |
PR00759 |
|
BASICPTASE |
IPR002223 |
PS00280 |
BPTI_KUNITZ_1 |
|
IPR002223 |
PS50279 |
|
BPTI_KUNITZ_2 |
IPR002223 |
SM00131 |
KU |
|
IPR002223 |
SSF57362 |
|
Prot_inh_Kunz-m |
IPR008154 |
PF02177 |
A4_EXTRA |
|
IPR008154 |
SM00006 |
|
A4_EXTRA |
IPR008154 |
SSF56491 |
A4_extra |
|
IPR008155 |
PR00203 |
|
AMYLOIDA4 |
IPR008155 |
PS00319 |
A4_EXTRA |
|
IPR008155 |
PS00320 |
|
A4_INTRA |
IPR013803 |
G3DSA:4.10.230.10 |
Beta‑amyloid peptide |
|
IPR013803 |
PF03494 |
Beta-APP |
|
IPR013803 |
PR00204 |
BETAAMYLOID |
|
Structural Features |
|
|
|
1aap |
1aap A,B |
|
|
1mwp |
1mwpA |
|
|
1owt |
1owtA |
|
|
1tkn |
1tknA |
|
|
2beq |
2beq A-E |
|
|
3.90.570.10.1 |
1mwpA0 |
|
|
4.10.230.10.1 |
1aml00 |
|
|
4.10.410.10.3 |
1aapA0 |
|
|
a.47.4.1 |
d1tkna_ |
|
|
d.170.2.1 |
d1mwpa_ |
|
|
d.230.3.1 |
d1owta_ |
|
|
g.8.1.1 |
d1aapa_ |
|
|
j.42.1.1 |
d1amb__ |
|
|
Structural Predictions |
|
|
|
MB_P05067 |
|
|
From the graphical match above, you can see that the signatures are all grouped into four InterPro entries for human amyloid-b precursor protein, APP. InterPro entries group together all the signatures that represent the same sequence found in the same set of proteins. These entries provide a family grouping of APP proteins, as well as subdividing APP into three constituent domains (the remaining domains are not currently represented by any signatures).
FAMILY Entries
Ø IPR008155: Amyloidogenic glycoprotein family, represented by three signatures: PR00203 (PRINTS), PS00319 (PROSITE), and PS00320 (PROSITE).
DOMAIN Entries
Ø IPR008154: N-terminal extracellular domain. Represented by three signatures: PF02177 (PFAM), SM00006 (SMART), and SSF56491 (SUPERFAMILY).
Ø IPR002223: Kunitz proteinases inhibitor I2 domain (which is found in several other protein families as well). Represented by eight signatures: G3DSA:4.10.410.10 (GENE3D), PD000222 (PRODOM), PF00014 (PFAM), PR00759 (PRINTS), PS00280 (PROSITE), PS50279 (PROSITE), SM00131 (SMART), and SSF57362 (SUPERFAMILY).
Ø IPR013803: b-amyloid peptide that is cleaved from the APP protein (prior to the C-terminal cytoplasmic domain). Represented by three signatures: G3DSA:4.10.230.10 (GENE3D), PF03494 (PFAM), and PR00204 (PRINTS).
The remaining fourteen entries in the table above give information on the known and predicted structure of this protein. The first thirteen entries present known structural data from the structural database PDB (green stripe) and the structural classification databases CATH (pink stripe) and SCOP (black stripe) (the names such as 1aapA0 are derived from the PDB entry upon which they are based, here PDB entry 1aap, chain A). There are five PDB entries that represent this protein: 1aap (chain A and B), 1mwp (chain A), 1owt (chain A), 1tkn (chain A), and 2beg (chains A to E). The CATH and SCOP entries provide a structural classification for each domain represented in the PDB data, and relate it to other structures in the PDB database. CATH has classified three of the PDB structures, while SCOP classifies all five:
Ø N-terminal extracellular heparin-binding domain is represented by 3.90.570.10.1 (CATH) and d.170.2.1 (SCOP), and has a disulphide-rich alpha+beta fold
Ø Extracellular copper-binding domain is represented by d.230.3.1 (SCOP), and has a 2-layer beta/alpha/beta fold
Ø Kunitz-type inhibitor domain is represented by 4.10.410.10.3 (CATH) and g.8.1.1 (SCOP), and has a disulphide-rich alpha+beta fold
Ø CAPPD extracellular domain is represented by a.47.4.1 (SCOP), and has an alpha-helical bundle fold
Ø Amyloid-b peptide is represented by 4.10.230.10.1 (CATH) and j.42.1.1 (SCOP), and has an irregular fold
It is interesting to note that two SCOP classifications subdivide the InterPro entry IPR008154 (N-terminal extracellular domain) into two structural subdomains: the heparin-binding subdomain and the copper-binding subdomain.
The
remaining entry is for the structural prediction, MB_P05067, from the homology model database
ModBase (yellow stripe). The homology
model provides information on the predicted structure for regions of the
proteins that have no known structure, here the region between the Kunitz-type
inhibitor domain and the CAPPD extracellular domain.
Structures
associated with APP can be viewed using AstexViewer®, which is linked from the
Match Table via the logo on the InterPro page (please note, there is no link directly from
this page to the AstexViewer®, therefore you need to go to the link on the InterPro page for P05067). The AstexViewer® displays the PDB structure with the specific
CATH or SCOP domain highlighted.
There are
structures available for various APP proteins in the Protein Data Bank
(PDB). A detailed description and
visualisation of the structural features of this protein can be found at the PDB ‘Molecule of the Month’.
The crystallographic structures of APP have provided insight into
several aspects of Alzheimer’s disease.